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Null mutations in Mc4r cause pronounced, dominantly inherited, monogenic obesity in rodents and humans, and are associated with increased food intake, decreased energy expenditure, and increased lean body and fat mass [56,57]
The level of CysSSH has also been estimated in M range and the level of both endogenous persulfides (CysSSH and GSSH) was dependent on the availability of sulfur compounds (e.g
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N., Riedl, C., Shah, S
