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Since these pathogen proteins and metabolites are often similarly structured to ones created by the human host, such molecular mimicry makes it difficult for the host to recognise the foreign pathogen (193), resulting in the hosts immune system becoming increasingly sluggish and more susceptible to acquiring other infectious agents

In addition to viral co-infection, antiCOVID19 therapies (i.e., azithromycin, nafamostat mesylate, and remdesivir) could activate various cell signaling pathways and trigger viral lytic reactivation 261

Meanwhile, mitochondrial function is maintained by a balance between fission and fusion, known as mitochondrial dynamics

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Retrieved from Millennium Physicians