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Neuropeptide substance P and the immune response

Further research indicates that this regulatory process is closely related to oxidative stress mediated by ROS and nitric oxide (NO) induced by inflammatory factors: ROS/NO promotes DMT1 expression and inhibits FPN1 function by upregulating iron regulatory protein 1 (IRP1) and hepcidin, ultimately forming a vicious cycle of iron overload-inflammation-iron retention [98]

Regulation of carbohydrate-responsive metabolic genes by histone acetylation and the acetylated histone reader BRD4 in the gene body region

, Watanabe, K

Additionally, OLES-25%HYT downregulated pro-inflammatory cytokines and the apoptotic gene Bax, while upregulating the anti-apoptotic gene Bcl2, demonstrating its intestinal anti-inflammatory, antioxidant, and anti-apoptotic effects in experimental ulcerative colitis models