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Up to 50% SOD deficiency in patients with supraventricular and ventricular arrhythmias demonstrates the involvement of oxidative stress in cardiac arrhythmic pathogenesis ( 2 - accumulation, affect H 2 O 2 concentration, misregulate many redox-sensitive pathways and may cause myocardial remodeling and failure ( 2 - ( 2 - levels in the vasculature (Yin

(iv) the prevention and/or treatment of cardiovascular disease, such as the delaying or reduction of the development of a major adverse cardiovascular event (MACE) selected from the group consisting of cardiovascular death, non-fatal myocardial infarction, non-fatal stroke, revascularisation, hospitalisation for unstable angina pectoris, and hospitalisation for heart failure

and, in part, by DFG RTG 2737 (STRESSistance)

doi: 10.1007/s00726-004-0114-4

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