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Exogenous hydrogen sulfide (H2S) was able to reverse the expression levels of Bax, Bcl-2, phosphorylated p38 MAPK, and P-selectin and significantly inhibited homocysteine-induced cellular ROS production, platelet activation, and NET formation, thereby protecting endothelial cells (275)

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Caspase-1 is the effector component of the NLRP3 inflammasome and is activated through proximity-induced autocatalytic activation upon recruitment to the NLRP3 inflammasome