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identified withanolide AC, a bioactive metabolite from Tubocapsicum anomalum , as a potent ferroptosis inducer that promotes GPX4 ubiquitination and autophagic degradation, suggesting it as a lead candidate for TNBC therapy [41]

This tripeptide represents the principal intracellular antioxidant defense system, maintaining redox homeostasis through its thiol (-SH) group that can donate electrons to neutralize reactive oxygen species (ROS) and reactive nitrogen species (RNS)

Cytokines like tumor necrosis factor (TNF) are the main means by which these activated cells foster a chronic inflammatory state and impact processes like neo-angiogenesis, epidermal hyperproliferation, differentiation, and apoptosis

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TABLE 2 Summary of HGF/c-Met signaling inhibitors in clinical trials