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On the other hand, mitochondria can effectively reduce ETC-derived ROS by activating uncoupling proteins, which lower the inner membrane potential a key driver of ROS production, thereby preserving cellular redox homeostasis [142]

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2.9 Construction of acute hyperuricemia models Healthy male BALB/c mice (20 g) were randomly assigned to four groups: Control, Model, Uricase, and RBC@SeMSN@Uri

while this effect is lost locally in the kidney after IL-10 neutralizing antibody treatment, suggesting that the protective effect of AT2R agonist C21 is mediated by IL-10 [25]