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JS: Writing original draft

Short-term MAPK inhibition exacerbates this resistance phenotype through compensatory ECM stiffening, creating self-reinforcing loops of mechanochemical signaling that maintain tumor-ECM coadaptation [140]

Mitochondrial dysfunction and antioxidant therapy in sepsis

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The main antigens expressed by medulloblastoma cells and/or by components of the tumor microenvironment will be considered as the molecular targets of antibodies, antibody derivatives, and chimeric antigen receptor effector cells to improve the conventional therapies