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Of note, pharmacologically induced PFC NMDAR hypofunction leads to increased oscillations [99] while genetic deletion of NMDAR1 subunits in pyramidal neurons increases oscillations, cell excitability and sEPSC frequency [100], similar to the present data

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[2] This correlation has led to the hypothesis that GHK depletion is not merely a marker of aging but a contributor to it, and that restoring GHK levels could partially reverse age-related tissue decline

Wu X, Yang Y, Dou Y, Ye J, Bian D, Wei Z

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