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A major limitation for translating NRF2-targeted strategies into clinical practice is the substantial genetic and molecular heterogeneity of HCC, which is due to diverse etiologies and distinct oncogenic alterations

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Curcumin exerts multitargeted neuroprotective actions, ranging from blocking the aggregation of toxic synuclein protein to activating prosurvival signaling pathways (e.g., PI3K/Akt and BDNF pathways) [48, 67]

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