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Some evidence supports aeroallergens in the etiology of EoE (Moawad et al., 2010), while other evidence refutes the theory (Lucendo et al., 2015)

Lipid hydroperoxides are substrates for Prx2 (Figure 5) [51] suggesting an anti-ferroptotic function as demonstrated in adipose-derived mesenchymal stem cells [151]

144 Of note, while the innate resistance of pancreatic carcinoma cells to CDDP and PARP1 inhibitors resolves simultaneously in response to HR-restoring BRCA2 mutations, 143 we have recently reported that NSCLC cells cultured for prolonged periods in the presence of subtoxic amounts of CDDP often (but not always) manifest a constitutive hyperactivation of PARP1, hence becoming susceptible to pharmacological or genetic PARP inhibition

Revealing binding sites for myeloperoxidase on the surface of human low density lipoproteins

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