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Unlike conventional compensatory pathway activation, KU-57788 exerts its effect on a novel target, DRP1, inducing mitochondrial hyper-fragmentation-mediated oxidative stress protection, which ultimately attenuates its own anticancer activity
12:e0175796
Otero M, Lago R, Gmez R, Lago F, Gomez-Reino JJ, Gualillo O
Where can capacity for SEL best be built in your institution to incentivize structural change and collaborative decision-making
J Neurochem 52: 11271134
