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Hormesis refers to a biphasic, dose-dependent phenomenon where low doses elicit beneficial cellular response, while high doses become detrimental to cell function or are toxic

These proinflammatory molecules also induce the above lesions through the nuclear factor-B (NF-B), Janus kinase/signal transducers and activators of transcription (JAK/STAT), and TGF-/Smad signaling pathways[99-101]