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Amyloid burden identifies neuropsychological phenotypes at increased risk of progression to Alzheimers disease in mild cognitive impairment patients

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91 AGEs, formed through the reactions of proteins and lipids with high glucose levels, crosslink extracellular matrix (ECM) proteins, inhibit ECM degradation by matrix metalloproteinases (MMPs) and increase cardiac stiffness, resulting in diastolic dysfunction

To facilitate the interpretation of results, we presented absolute effects for all outcomes