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It functions by interacting with specific high-affinity ligands, triggering a cascade of biological responses that researchers are eager to quantify

However, during high-fat diet (HFD) feeding, obesity and insulin resistance, the accumulation of medium-chain and long-chain acylcarnitines due to incomplete -oxidation may negatively regulate the expression of nuclear-encoded mitochondrial genes, further reducing mitochondrial number and function and creating a vicious metabolic cycle [33]

For these patients, an alternative methodology can be adopted to assess the presence and relative abundance of circulating endogenous factors that may inhibit LPL activity, leading to HTG