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These observations have generated interest in astrocyte glutamate handling as a potential ischemic therapeutic target, since upregulated expression or activity of glutamate transporters or inhibition of volume-regulated anion channels may decrease glutamate excitotoxicity (Rossi et al., 2007)

The increase in total iron concentration with aging could be caused by several factors, including increased permeability of the BBB [52, 53], the redistribution of iron within the brain and changes in iron homeostasis

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