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This results in increased proteasomal degradation of oxidatively damaged proteins as well as p62, thereby facilitating ATG-lysosomal degradation of proteins and dysfunctional organelles, such as the mitochondria (Levonen et al., 2014)
L, Zhou, Tan F, Zhang X, Li Y, and Yin W
You acknowledge that her disease is not well controlled and discuss the potential benefit and adverse events of increasing treatment doses and d
