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also plays a vital role in fatty acid synthesis and energy production

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While these data indicate that GSK3 is a key contributor to the GC, the disparity between data obtained after mTORC2 versus GSK3 inactivation raises the possibility that an input to GSK3 other than PI3Ksuch as an input from the WNT pathway [192]is a more important physiological factor than PI3K signaling

When efferocytosis is impaired, and adipocyte apoptosis increases in morbid obesity, metabolic syndrome, and T2DM, apoptotic bodies undergo secondary necrosis, releasing DAMPs that amplify local inflammation and reinforce a self-sustaining loop between adipose remodeling, immune activation, and insulin resistance [967,968,969,970]

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