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101 LutzH
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Role of endoplasmic reticulum stress in the male reproductive system
Cholesterol upregulates the expression of inhibitory immune checkpoint receptors such as PD-1, TIM-3, and LAG-3 on CD8 + T cells, and enhances the transcriptional activity of stress-responsive transcription factors, which drives exhaustion-related gene programs
and optimal dose fractionation schedules are also developed to allow better cumulative damages to the tumor and adequate repairing of normal tissues 2,3,4,5
