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Importantly, suppression of HSP70 reduced ER retention and degradation of the mutant protein and promoted transport to the Golgi and cell surface, suggesting that HSP70 could be a viable pharmacological target to increase proper localization of ATP7B-H1069Q

11G), while decreased the levels of SOD2, CAT (Fig

Therefore, the findings are not completely consistent across species, likely because humans are long-lived while mice have much shorter lifespans, resulting in different disease time courses and inflammatory milieu

Science 292 , 20772080 (2001)

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