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Preclinical studies have revealed that NAC inhibits several profibrotic mechanisms in a number of experimental models [39]

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We set out to develop biochemical biomarkers of target engagement (a direct measure of drug action) to measure the effects of therapies targeting oxidative stress, including edaravone and nuclear factor erythroid 2-related factor 2 (NRF2) activators [3], which show promise as disease-modifying agents for neurodegenerative diseases

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