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The Thalassaemia Syndromes (4th ed.)
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However, these initial events may be transient and insufficient to induce hepatocellular death and hence overt liver injury
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Inflammatory M1 phenotype macrophages exhibit increased oxidative stress, impaired cholesterol efflux and enhanced cytokine/chemokine secretion, leading to more LDL/remnant oxidation, endothelial cell activation, monocyte recruitment, and foam cell formation (54-59)
