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Since the anion transport of AtCLCa depends on the H + -coupled anti-transport and the proton glutamate mutant E270A abolished both H + and anion transport 29 , we suggest that the binding of PI(3,5)P 2 to the dimer interfaces of the TMD may occupy the cytoplasmic outlet of H + transport pathway, therefore attenuate the AtCLCa activity (Fig
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The Journal of biological chemistry 2011
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