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Thymosin alpha-1 is often considered the most logical starting point due to its well-established safety profile, its direct immune-modulatory mechanism, and the finding that lupus patients tend to have lower endogenous levels

While they are often discussed together, they operate in different ways and serve distinct roles in an optimized healing protocol

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pro-apoptotic pathways, including the activation of caspases and prolonged c-Jun NH2 terminal kinase (JNK) signaling, are antagonized by various NF-B target genes 28