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Taken together, decrease in expression of inflammatory markers such as pro-inflammatory cytokines and chemokines and infiltration of pro-inflammatory macrophages in adipose tissue of Cpt1b m/ mice indicates that inflammatory status is improved in adipose tissue despite the elevated levels of lipids in circulation

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The mechanisms are complementary, making this combination theoretically rational

Individuals with less adipose tissue have fewer fat cells available for lipolysis, and the substrate for the enzyme cascade diminishes

Taken together, Cpt1b m/ muscle has more type 1 fibers and less type 2b fibers, however, it appears that fiber type distribution or fiber-switching does not affect expression of inflammatory markers or immune cell infiltration in skeletal muscle of Cpt1b m/ mice