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The frequent occurrence of demodecidosis in patients with immunodeficiency and vascular insufficiency suggests that an abnormal vascularized surface as well as a compromised patient immune status along with genetic predisposition might be suspected as factors promoting the proliferation of demodex in rosacea [61,62,63]
The sophisticated glycosylation end-product-specific receptor controls macrophage inflammatory responses by HMGB1 (101)
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Comparison with the Effects of GHRH, TRH and hCRH, Peptides, Volume 18, Issue 6, 1997, Pages 885-891, ISSN 0196-9781, Chihara K, Shimatsu A, Hizuka N, Tanaka T, Seino Y, Katofor Y
Pharmacological Research , 193 , 106812
