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Abnormalities in the mitochondrial electron transport chain (ETC), combined with impaired antioxidant defenses, lead to excessive accumulation of reactive oxygen species (ROS), which damage lipids, proteins, and mitochondrial DNA, ultimately promoting tubular apoptosis, interstitial fibrosis, and glomerulosclerosis (26, 27)

Importantly, glutathione deficiency was more pronounced in dogs that did not survive to discharge from the hospital, compared to survivors

L.TianS.et al (2007)

Postural tachycardia syndrome (POTS) J

These findings indicate that inhibiting ferroptosis in foam cells is essential for treating AS (Su et al., 2021)