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doi: 10.1016/j.jtos.2024.12.001
The peptide is then cleaved to produce mature and enzymatically active proteins that play a key role in mitochondria [15,21]
This dual "energy depletion-oxidative stress" insult forms a self-amplifying loop: lipid peroxides generated by ferroptosis further damage mitochondrial membrane integrity, while IL-1 released by pyroptosis exacerbates neuronal mitochondrial dysfunction through paracrine effects [23,24]
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