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Basketter, D., Darlenski, R

Co-cultures of glioma and neuronal cells demonstrated that the released l-Glu was sufficient to induce excitotoxic damage and that the neuronal loss could be reduced by either by preventing its efflux through Sx c - (4- S -CPG or SSZ) or inhibiting its action at NMDA receptors (MK-801)

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Samples were resuspended in 2% acetonitrile (ACN), 0.05%TFA and peptide concentrations were estimated with 205 nm absorbance readings (Nanodrop, Thermo Fischer)

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