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In particular, HT appears to act by modulating the NF-B signaling pathway, the consequent release of pro-inflammatory cytokines, and the expression of molecules downstream of the inflammatory cascade, such as COX-2 and iNOS [32,91,93,94,95,96,97]
Placentas from pregnancies with FGR have a significantly decreased expression of genes involved in mitochondrial function and oxidative phosphorylation but higher markers of OS [47]
doi: 10.1002/anie.201007871
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