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doi: 10.1038/s41467-019-10991-7

(2008) described the discovery and characterization of the tissue-protective EPO receptor (now called the innate repair receptor) and demonstrated that engineered EPO derivatives lacking erythropoietic activity retained cytoprotective signaling, laying the foundation for ARA-290 development [2]

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This involves randomly permuting the elements in one of the two vectors and calculating the correlation coefficient for each permutation

Mechanistic Rationale for Vitamin D, C, and E in Endometriosis The pathophysiological features summarized in Section 1.2, namely dysregulated estrogen signaling, progesterone resistance, chronic inflammation, oxidative stress, and immune dysfunction, provide a direct rationale for considering vitamins D, C, and E as adjunctive therapies in endometriosis
