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Furthermore, suppressing lipid-associated ROS production in neutrophils by LPX-1 treatment in MRL/lpr mice effectively attenuated disease progression, decreased production of autoantibodies and various inflammatory cytokines, increased serum C3 complement levels, and reduced the severity of splenomegaly, lymphadenopathy, and lupus nephritis
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All this reveals the strong impact that HO-1 elicits in the context of immune-mediated diseases, as well as its centrality for the maintenance of immune homeostasis
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