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LC and its derivatives have a favorable regulatory effect on mitochondrial diseases [4]
Trimethylamine-N-Oxide instigates NLRP3 inflammasome activation and endothelial dysfunction

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This study evaluated the role of prophylactic ALC administration on reducing the secondary injury mechanisms of axonal neurodegeneration, reactive astrogliosis and microgliosis, inflammation and glutamate excitotoxicity in the hippocampus and cortex resulting from rmTBI
