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A recent study looked at 13 research papers with 4275 patients

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First, hyperhomocysteinemia detected only in the fasting state (approximately 23% of patients), which might mainly result from an inhibition of the homocysteine remethylation pathway (folate, cobalamin, or functional methionine synthase deficiencies or thermolabile MTHFR mutation), and which increases the cardiovascular risk 1.6-fold

Chhipa, N

This is relevant, as platelets are the first cells that arrive at a site of injury to release various factors that initiate the repair process