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Healing and clinical improvement can not happen when this occurs
& Allen, R
Transgenic mice in which GCLC expression is abolished in hepatocytes have 58% of normal hepatic GSH levels and manifest liver pathologies characteristic of the various clinical stages of fatty liver disease 8,9
AGS cellular models were associated with accumulation of glutathione (GSH), a ROS scavenger indicative of oxidative stress 37 , and TCA feeding metabolite glutamate (GLU) (figure S7F right panel )
