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Growth hormone (GH)-transgenic insulin-like growth factor 1 (IGF1)-deficient mice allow dissociation of excess GH and IGF1 effects on glomerular and tubular growth

A review of the role of reactive oxygen and nitrogen species in alcohol-induced mitochondrial dysfunction

We used the model to study the extent of liver damage caused by overdoses or by chronic use of therapeutic doses, and the effects of polymorphisms in glucoronidation enzymes

Nefrologia 35:304321

doi: 10.1038/msb.2012.20 Supplementary Information Rights and permissions This is an openaccess article distributed under the terms of the Creative Commons Attribution License, which permits distribution, and reproduction in any medium, provided the original author and source are credited