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In addition, after DNA damage caused by OS, DNA repair mechanisms in glioma cells are activated to repair damaged DNA and exert indirect antioxidant effects, such as direct repair (5961), base excision repair (BER) (62), mismatch repair (MMR) (63, 64), and nucleotide excision repair (NER) (65, 66)
doi: 10.1073/pnas.0507865102
Critically, the soluble oligomeric form of -synuclein is now widely recognized as the pathogenic species in PD 45
Adaptation of orientation vectors of otolith-related central vestibular neurons to gravity
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