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To explore the in vivo metabolic effects caused by chronic stimulation of a G s -linked GPCR in SKM, we generated mice that expressed a G s -coupled DREADD (GsD) 32,33,34,35,36 under the transcriptional control of the SKM-specific human -skeletal actin (HSA) promoter

Similar content being viewed by others Background Kawasaki disease (KD) is a medium vessel vasculitis of undetermined etiology usually affecting children below 5 years [1,2,3]

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