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Three main hypotheses were proposed to explain how riboflavin deficiency can lead to low parasitaemia: haemolysis, inhibition of reticulocytosis, and oxidative stress

In HL-60 leukemia cancer cell lines, quercetin treatment induced FasL expression that triggered extrinsic apoptotic pathway, protein activation and conformational changes, and activation of ERK and JNK signaling pathways

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Impaired fasting glucose tolerance in first-episode, drug-naive patients with schizophrenia