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Abstract Oxidative stress, disrupted copper homeostasis, and neuroinflammation due to overproduction of proinflammatory cytokines are considered leading causative factors in development of age-associated neurodegenerative conditions

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We conducted titration experiments on the complex of 15 N-labeled p53 TAD (13-61) and unlabeled FOXO4 FHD with increasing concentrations of p53 TAD PEP

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