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Secondly, we monitored glucagon granule exocytosis in cells by tracking the trafficking of NPY-mCherry, which is driven by the pre-proglucagon promoter and serves as a well-established secretory granule marker 19 (Supplementary Fig
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One proposed solution is to administer dopamine directly to the brain, bypassing the need for its enzymatic conversion from levodopa

This suggests that quenching in absorbance of BSA occurred mainly due to TMAO-BSA conjugate formation [44]