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Ultimately, iron overload may promote the progression of OA through a cascade mechanism that disrupts intestinal homeostasis and exacerbates systemic inflammation

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These substances partially inhibit oxidative stress reactions, enhance endogenous antioxidant production, counteract hypoxia and stress from ischemic damage, and restore balance between endogenous antioxidants and prooxidants (36, 37)

Mitochondrial dysfunction, impaired axonal transport, and neuroinflammation are key contributors to oxidative stress in ALS [169]