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Animal models confirm TMAOs pathogenic role: reducing plasma TMAO slows CKD progression (138), while TMAO accelerates diabetic nephropathy by activating NLRP3 inflammasome, promoting IL-1/IL-18 release, and worsening renal dysfunction/fibrosis (139)
Briles, E
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It is therefore proposed that in such circumstance, cellular checkpoint processes may instigate the formation of heterochromatin (Hahn et al., 2010)
