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FPN activity is controlled by another protein produced in the liver called hepcidin, which reduces iron translocation by degrading FPN in the basement membranes of duodenocytes, macrophages, and hepatocytes, thereby lowering serum iron levels [18]
Some of the neuro-protective effects seen with this drug are secondary to its anti-inflammatory and antioxidant actions and appear to rely on the modulation of cellular metabolism
Il aide ainsi permettre l'change d'nergie (donc d'lectron) dans la cellule
doi: 10.1039/c9fo01075k
Ultimately, activation of matrix metalloproteinases degrades collagen matrices and promotes smooth muscle cell apoptosis, leading to plaque destabilization [322]
