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Lemberger T, Desvergne B, Wahli W: Peroxisome proliferator activated receptors: a nuclear receptor signaling pathway in lipid physiology

Instead of the old term of non-alcoholic fatty liver disease (NAFLD), a more reasonable broader substitute, metabolic dysfunction-associated steatotic liver disease (MASLD), became the spotlight because it can cover huge and diverse causes of steatosis under its umbrella (Rinella et al

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While gut bacteria metabolize LC into TMA, which is then converted to TMAO in the liver, these derivatives have distinct metabolic pathways and bioavailability, which may influence the amount of TMAO levels [32, 48, 49]

Furthermore, the transporter for T4 is much more energy dependent (it requires more energy) than the transporter for T3 (see Figure 1)