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The inhibitory effects may also be tissue dependent as orally administered acetate increased acetylation and inhibit the activity and expression of HDAC2 in rodent brain (Soliman and Rosenberger, 2011)
Ylmaz et al
This leaves the thyroid cells incapable of performing their physiological job, while also being incapable of repairing damage caused by oxidative stress
Journal of Medicinal Chemistry, 57 (22), 9220
