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Travis WD, Dacic S, Wistuba I, Sholl L, Adusumilli P, Bubendorf L, Bunn P, Cascone T, Chaft J, Chen G, Chou TY, Cooper W, Erasmus JJ, Ferreira CG, Goo JM, Heymach J, Hirsch FR, Horinouchi H, Kerr K, Kris M, Jain D, Kim YT, Lopez-Rios F, Lu S, Mitsudomi T, Moreira A, Motoi N, Nicholson AG, Oliveira R, Papotti M, Pastorino U, Paz-Ares L, Pelosi G, Poleri C, Provencio M, Roden AC, Scagliotti G, Swisher SG , Thunnissen E, Tsao MS, Vansteenkiste J, Weder W, Yatabe Y

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Apoptotic bodies derived from the damaged hepatic cells can further activate Kupffer cells to secrete transforming growth factor beta 1, endothelial growth factor and platelet-derived growth factor, which can promote the transformation of activated hepatic stellate cells into myofibroblasts [19]

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The supply of NADPH is essential to regenerate GSH and dictates the rate of H 2 O 2 reduction by Gpx while keeping the reduced status of mitochondria