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There is mounting evidence that, irrespective of eosinophilic inflammation, IL-13 mimics many of the characteristics of asthma in mice, including AHR and mucus hypersecretion, and potently stimulates the release of eotaxin from airway epithelial cells [191,192]

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Spectrophotometry, 2020a

Together with existing docking and pharmacological studies, these findings support a general pharmacophore in which a conserved basic center, C15/C20 hydrophobic substituents, and configuration-dependent orientation of the indole/oxindole core jointly dictate MOR affinity, subtype selectivity, and potentially signaling bias (Figure 11)

held an FPI fellowship from the Spanish Ministerio de Educacion y Ciencia (BES-2014-069236)