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Description
This dichotomy suggests that autophagy dysfunction may contribute to leukemia development, with an imbalance in its regulatory network accelerating the emergence of malignant phenotypes (39)
For biochemical assays, each 0.5 g sample of leaf material was homogenized in 3 mL 25 mM HEPES buffer (pH 7.8) containing 0.2 mM EDTA and 2% (w/v) polyvinylpyrrolidon
Oncogene 28, 12611273
SIRT1/Atg5/autophagy are involved in the antiatherosclerosis effects of ursolic acid
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