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Abstract The proinflammatory (N1) polarization of bone marrow (BM) neutrophils, driven by central immune remodeling in response to peripheral inflammation, plays a critical role in propagating localized inflammatory conditions, such as periodontitis, to systemic levels

Overexpression of AR was found to promote liver inflammation and HCC development via activation of AKT/mTOR signaling by interaction with the kinase domain of AKT1 [138]

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& Salamonsen, L